Before everything, I believe that one very important thing should be mentioned.
Peptides that have disulfide bridges must be cultivated on recombinant bacteria, and protein hormones such as insulin, IGF-1, HGH or Frag 176- 191 contain 3 or more disulfide bridges. They are the key element of the spatial arrangement for such molecules (the so-called protein conformation - protein hormones
inappropriately conformed have little or no activity at best at the target receptor. Therefore, quite often you can find overdosage for these reasons. If you don't feel 100 mcg Frag dear reader, it just wasn't well done.
A peptide is a short chain of amino acids linked by a bond. The chain typically has 50 amino acids or less with an amino carboxyl terminus. Peptides occur naturally and are synthesized, often confused with proteins, perhaps because both are made of amino acids. Unfortunately, there is one significant difference
- proteins have more than 50 amino acids. Peptides are used to produce hormones that are protein-based, such as growth
hormone, and there are many types of peptides, including polymers, oligopeptides and neuropeptides.

Peptide Dosing Reference Table

We are interested in all those that promote hGH release, and some are said to increase testosterone production as well as their ability to accelerate regeneration. They help to transport oxygen to the muscle cells in the right amount and have a positive effect on efficiency. Another reason to make friends with peptides is their ability to burn fat.
Access to peptides is not as impaired yet as to steroids, so we will mainly focus on the quality and purity of the agents. We divide peptides into groups:
GHRH, i.e. growth hormone releasing hormones.
The name itself should trigger logical thinking - they will stimulate the release of growth hormone. The duration of action will depend on the type of peptide used. Please note. that peptides are only effective up to a certain level. The supersaturation dose varies from person to person, and exceeding it has no measurable advantages and will not improve the amount of forced HGH burst. These types of peptides include:
MOD – GRF (1 - 29)
Half-life of about half an hour, has a positive effect on muscle tissue growth, regeneration, slows down the aging process, also known as CJC - 1295 without DAC
CJC-1295 z DAC
The main difference between CJC- 1295 DAC and MOD GRF 1 - 29 (CJC-
1295 without DAC) is the growth hormone pulse duration. While MOD GRF 1 - 29 has a fairly short half-life and increases growth hormone secretion only for a short time after injection, CJC-1295 DAC has a fairly long half-life of several days and
increases the sustained release of growth hormone by the pituitary gland. It has two tasks - to permanently and pulsating (naturally) increase the release of growth hormone by the pituitary gland, and thus to keep the IGF-1 level at an elevated level. The optimal levels of these hormones for
a person will result in a much better breakdown and burning of adipose tissue, as well as complement the steroid-initiated excessive protein synthesis - i.e. the hypertrophy of new muscle fibers, which is most useful during hormone therapy. There is a study to investigate the pharmacokinetic profile, pharmacodynamic effects, and safety of CJC-1295, a long-acting        GHRH    analog. (https://academic.oup.com/jcem/article/91/3/799/2843281
)
After a single dose of CJC-1295, there was a dose- dependent increase in mean blood levels of hGH two to 10 times for 6 or more days and in mean IGF-I plasma levels by
1.5 to 3 times for 9-11 days . The estimated half-life of CJC- 1295 was 5.8 - 8.1 d. After multiple doses of CJC-1295, mean IGF-I levels were above baseline for up to 28 days. No serious adverse events have been reported.
The conclusion of the study is as follows:
Subcutaneous administration of CJC-1295 resulted in a prolonged, dose-dependent elevation of GH and IGF-I levels in healthy adult humans and was safe and relatively well tolerated, especially at doses of 30 or 60 mcg / kg. After multiple doses, there was evidence of a cumulative effect. These data support the potential utility of CJC-1295 as a therapeutic (or bodybuilding substance ...?)
The study design consisted of two randomized, placebo- controlled, double-blind, dose escalation trials of 28 days and 49 days. The study was conducted in two research centers. SFBC International - Miami Kendle International BV- Utrecht. Healthy people (men and women) aged 21-61 years were examined.
Study 1 evaluated four consecutive groups at different doses. These dose levels were 30 mcg / kg (n = 6, five active and one placebo), 60 mcg / kg (n = 6, five ak325; '[2,178 actives and one placebo), 125 mcg / kg (n = 6, five active and one placebo) and 250 μg / kg (n = 6, five active and one placebo). An additional group of 18 subjects (15 active subjects and 3 placebo) then received 125 μg / kg.
The mean maximum GH concentrations were 6.6, 9.6,
9.9 and 13.3 ng / ml in the 30, 60, 125 and 250 µg / kg groups; mean AUC (this parameter informs about the total amount of drug that was absorbed into the body) were respectively 758, 969, 977 and 1370 ng / ml, and thus also levels of igf1 after repeated use of cjc increased and lasted up to 28 days and their maximum concentration was observed on the eighth day.
Side effects that have been observed:
skin irritation, erythema, induration, pain or itching occurred transiently (up to several hours) in approximately
70% of patients receiving CJC-1295 and rarely in patients receiving placebo. Injection site reactions were more severe and / or prolonged at higher doses, with residual induration lasting up to 5 days. No local reactions exceeded 10 cm in diameter and all spontaneously separated. Transient injection site urticaria rashes occurred in nearly 30% of patients and were not dose related. Other adverse reactions reported in actively treated patients were headache (63%), diarrhea (43%), and systemic vasodilating reactions (redness, warmth, and transient hypotension; 30%); all were more frequent at the higher doses (125 or 250 mcg / kg)
HGH      Fragment           176-
191
As the name suggests, it is a fragment of growth hormone, and in particular a modified form of amino acids 176-191. Used mainly for fat burning - here it works much more efficiently and faster than HGH itself and is, above all, a much cheaper alternative. We will also experience anti-aging benefits and an increase in IGF levels -
Dosage: 100 mcg of good Fraga is perfectly sufficient (2 - 3 times a day max).
If you don't feel 100 mcg Frag then you don't have the real Frag.
If you feel an increased appetite after Frag, then you don't have a real Frag.
Sermorelin
Structurally similar to CJC - 1295 commonly used for anti- aging purposes, but also useful for building muscle. It contains 29 out of 44 amino acids that form growth hormone releasing hormones. This peptide is very effective in improving HGH levels as shown in studies. A chain of 1-29 amino acids has been
observed to be mainly responsible for the ability of GHRH to stimulate the release of growth hormone by the pituitary gland. However, Sermorelin has a very short half-life of about 10 minutes or less. One possibility that is gaining popularity is the use of growth hormone secretagogues to promote pituitary health and function during aging. An example of such molecules is growth hormone releasing factor 1-29 NH2 acetate or Sermorelin         (Miriam              et           al.           2001).
Some of them have been shown to be effective in improving the physical fitness of the elderly (Fahy 2006). However, it is unlikely that they will be sold for the next few years, and maybe a dozen or so in Poland: P. On the other hand, Sermorelin, an analog of the naturally occurring growth hormone releasing hormone (GHRH) that declines during aging, may now be a more direct and better alternative to hGH. for HRT in the aging process (Russell-Aulet et al. 2001). The molecule was manufactured and marketed for many years as an alternative to hGH for use in children with growth retardation, but it could not compete with hGH and was discontinued by the manufacturer as a therapeutic unit. Paradoxically, Sermorelin has failed as a growth stimulant in children for the same reason that it is a better alternative to HRT in aging adults. Children with growth deficiency need higher doses of growth hormone than can be achieved by stimulating the production of their own hormone, (maybe the person who went through this will write the doses
that were assigned, we saw you recently (mad respect bro, will be with CB an excellent doctor) while the beneficial effects sermorelin on pituitary gland function and simulation of growth hormone secretory dynamics in aging adults have little effect on growth rate in children.Unlike exogenous hGH, which produces bioactive IGF-1 from the liver, Sermorelin stimulates the pituitary gland by binding to certain receptors to increase endogenous hgh production and secretion Since Sermorelin increases endogenous hgh by stimulating the pituitary gland, it has some physiological and clinical advantages over hGH which include:
The effects are regulated by a negative feedback loop involving the inhibitory neurohormone somastatin, so that unlike the administration of exogenous hGH, overdosing of endogenous  hGH  is  difficult,  if  not  impossible.
Due to the interactive effects of Sermorelin and Somatostaine, the release of hGH by the pituitary gland is episodic or intermittent and not constant as with hGH.
Tachphylaxis is avoided because Sermorelin-induced pituitary hGH release stimulates a more normal physiology.Sermorelin stimulates the transcription of the hGH messenger pituitary RNA gene, increasing the pituitary reserve and  thus  preserving  more  of  the  growth  hormone
neuroendocrine axis which first fails during aging (Walker et al. 1994).
Pituitary recurrence resulting from Sermorelin helps to slow down the cascade of pituitary failure that occurs during aging, and therefore, if we know what it is in adolescence and early 20s, we can guess the rest (Villalobos et al. 1997)
Melanotan
Melanotan II is a preparation containing synthetic analogues of the melanotropic hormone,
i.e. the alpha-melanotropic hormone. In the body, it is produced in the pituitary gland, from where it acts on pigment- containing cells, i.e. melanin. The purpose of Melanotan II is to stimulate the melanocytes, that is, the skin's pigment cells, to produce even more  pigment.
The obtained tan is more durable
than the one obtained on a tanning bed or with the help of self-
tanning agents, and the effect appears after just a few injections. Melanotan itself was first tested by American scientists as a drug for diseases related to photosensitivity. However, during the research it turned out that the preparation can increase libido for a short time and allow you to control your weight. The very operation of Melanotan II is very simple. The peptide is designed to stimulate the body to sunbathe with minimal exposure to sunlight. Additionally, by using this peptide, our skin regenerates much faster and any damage heals faster. Ultimately, the tan we get is very permanent. Naturally tanned skin maintains the tan for several weeks. In this case, we can enjoy darker skin for up to several months. Therefore, we are able to tan the body for the entire winter season. It is also an ideal solution for people who have a very fair complexion and sunbathing means for them to burn their body and get a red tan at best. The peptide itself also has another advantage that men will surely appreciate. It can significantly increase the tendency to get an erection. To achieve this goal, you should have an injection six hours before your planned intercourse. The operation itself can take up to twelve hours.
Depending on the degree of tolerance of each person, I suggest gradually increasing the dose - too high a dose may cause joint pain, bone pain, muscle tightness, headaches, and irritability. A very high libido and symptoms resembling a strong flu are certainly not pleasant.
Epithalon
At the end of each chromosome there are several thousand copies of a certain DNA sequence made up of six pairs of telomeres. In the course of each cell division - the chromosomes of somatic cells lose about 200 pairs of telomeres.
According to the telomere theory, the length of telomere limits the life span of somatic cells and thus
of the whole human body (Finkel 1998).
Age- related shutdown of oestrus function and frequency of chromosomal aberrations in bone marrow cells slowed when mice were administered Epitalon. The viability of the mice treated with Epitalon was increased by 13.3% of the last 10% of the mice that survived the experiment, and the viability of mice in general was increased by 12.3% compared to the control mice. It was noted that the peptide had no effect on cancer cells to multiply them, but inhibited the development of leukemia, suggesting a protective effect of Epitalon on geroprotectors, and without side effects during long-term administration of the peptide to mice.
A geroprotector is a senotherapist whose goal is to influence the root cause of aging and age-related diseases, thereby
prolonging life. Some possible geroprotectors include melatonin, carnosine, metformin, nicotinamide mononucleotide (NMN). For centuries, mankind has searched for the Fountain of Youth; the proverbial source of eternal life. Herodotus wrote about the spring that gave the water of youth to all who bathed in it. Juan Ponce de Leon searched for him in South Florida centuries later but did not find him. Man's search for such a fountain was unsuccessful until Dr. Vladimir Khavinson discovered Epitalon in the 1980s. The fountain turned out to be a peptide produced by the pineal gland.
Prevents DNA from breaking down during any cell division. Each cell division results in a slightly shorter telomere length and ultimately the cell cannot divide any further. This is called the Hayflick Limit, following the discovery of Dr. Leonard Hayflick - cells have a limited number of times they can divide. In mammals, telomeres are protected from shortening until the onset of sexual maturity. They then begin to shorten with each cell division, ultimately making it impossible to divide further to replace worn out, damaged or diseased cells. The telomerase enzyme stimulates the elongation of the telomere.
The pineal gland produces a hormone called epitalamine, which tells cells to make telomerase, which in turn results in longer telomeres in our DNA. Pineal gland functionality declines with age and is partly responsible for age-related diseases. Dr. Khavinson found that the introduction of epitalamine in mammals reversed age-related diseases and reversed signs of
aging. He was able to take the female mice that were no longer fertile, give them epitalamine, and after about two weeks of treatment, the mice became fertile again, became pregnant, and gave birth to pups. He showed that Epitalon induces telomerase activity in human somatic cells, proving that peptides elongated telomeres. A synthetic version of epitalamine was patented by Dr. Khavinson and named "Epitalon". It was approved for general use in the Soviet Union in 1990 and has been used in gerontology there ever since.
Almost all of the studies have been conducted by Dr. Khavinson and his colleagues. The results of his research are surprising: for example, the use of epitalamine reduced mortality in humans at the age of 1.8 times over 6 years of follow-up. Because Epitalon is patented in the west, it is not clinically tested in humans. A tiny peptide composed of the four amino acids ALA-GLU-ASP-GLY and can be administered subcutaneously, intranasally or intramuscularly, although it works best subcutaneously or intramuscularly, optimally 2-3 times a day for 10 - 20 days in doses of 5 - 10 mg per dose once every 6 to 12 months, but may also be given more frequently. No negative side effects have been reported in more than 100 studies of epithelial and clinical use in Russia since 1990. It works mainly on the endocrine system, but has an effect on our entire body. It improves the senses such as taste, smell, vision, digestion and sleep. All these functions are related to the
autonomic nervous system and the endocrine system. Epitalon has been shown to restore the normal production of melatonin in aging monkeys as well as restoring the normal circadian rhythm with the production of cortisol, resulting in better night sleep.
The next study involved 266 elderly people and lasted over a period of 6-8 years, where the bioregulators epitalon and thymalin were administered during the first 3 years of the experiment.
Observed: the ability of bioregulators to normalize the basic functions of the human body, such as: cardiovascular, endocrine, immune and nervous systems, homeostasis and metabolism. The restoration of homeostasis was accompanied by a 2.0 - 2.4 - fold decrease in the incidence of acute respiratory disease, a reduced incidence of clinical symptoms of ischemic heart disease, hypertension, deformity, arthrosis and osteoporosis compared to control. Such a significant improvement in the health of patients treated with the peptide correlated with reduced mortality during follow-up:
2.0 - 2.1 times in the thymalin group;
1.6 - 1.8 times in the epitalamine treatment group;
2.5 times in patients treated with thymalin and epitalamine
compared to controls.
A separate group of patients was treated with thymalin in combination with epitalamine annually for 6 years, and their mortality decreased 4.1 times compared to the control group.
The obtained data confirmed the high geroprotective efficacy of thymalin and epitalamine and the desirability of their use in medicine and social care in order to maintain health and age- related pathological prophylaxis in people over 60 years of age in order to extend their lives. ● ¨"
GHRP, or Growth Hormone Releasing Peptides (growth hormone releasing peptides)
Unlike GHRH, when giving everyone in the compounds listed below, we do not need to target the hours of our natural HGH secretion pulse. Growth hormone releasing peptides will cause an immediate burst of growth hormone each time they are given. The following compounds are available:
GHRP-6
It strongly promotes the release of HGH, helps fight inflammation, accelerates regeneration and fat burning. Not
only does it stimulate the pituitary gland to release hGH - it also inhibits Somastatin.
GHRP-2
Stronger though to a small extent than GHRP-6 when used for similar purposes but does not induce an appetite attack as strong as the previous one (it is easy to recognize the differences between them) and does not desensitize the receptors as much as GHRP-6 when taken long-term. Much more useful for gaining pure muscle mass than its predecessor.
Ipamorelin
Like GHRP-6, they stimulate the pituitary gland and inhibit Somatostatin, but like the above - it does not stimulate the appetite, does not increase prolactin or cortisol. It is not a strong stimulant of hGH secretion, but it is definitely worth attention for both men and women.
Haxarelin
Stronger when it comes to the release of hGH from GHRP-2 and 6, anti-aging, burns adipose tissue, on this occasion, stronger than the above-mentioned, you should take breaks from it, because it has a higher desensitization potential, regardless of the dose and duration of use.
Insulin-like growth factors
A class of peptides that works in a similar way to insulin, as the name suggests. They work locally and are available in several variants:
IGF-1 LR3
It is the most popular variant of IGF-1 on the market today. IGF-1 LR3 contains 83 amino acids. That is, it has an additional 13 amino acids to the standard insulin-like growth factor-1 sequence. This polypeptide has features that make it much more potent than normal IGF-1. It has a longer half- life of up to 30 hours, compared to 15 hours. In addition to satellite cell growth, IGF-1 LR3 helps burn fat, regenerate faster, and slow down aging. IGF1-LR3 is a modified version of insulin-like growth factor-1. The full name of the peptide is insulin-like long growth factor-1 arginine 3. All IGF-1
derivatives play a significant role in cell division, cell proliferation and communication between cells. Although it has a similar effect, IGF-1 LR3 does not adhere to IGF- binding proteins as tightly as IGF-1. This causes IGF1-LR3 to stay in the bloodstream 120 times longer than IGF-1. IGF1- LR3 gains an extended half-life as a result of structural changes. The peptide is made by adding 13 amino acids to the N-terminal end of IGF-1 and by converting the glutamic acid at position 3 of IGF-1 to an arginine residue. Like IGF-1, IGF1-LR3 gives a signal for cell division and proliferation. Its main activity affects connective tissues such as muscles and bones, but it also promotes cell division in the liver, kidneys, nerves, skin, lungs and blood.
As a result, IGF1-LR3 stays in the bloodstream for much longer IGF-1 and the same dose of long IGF-1 is about 3 times stronger in terms of cell activation, and in the case of IGF1-LR3 muscles, it does not cause muscle cell growth, but increases the total number of cells muscle.
IGF1-LR3 increases fat metabolism indirectly through binding to both the IGF-1R and the insulin receptor. These actions increase the uptake of glucose from the blood by the cells of the muscles, nerves and liver. This causes a general drop in blood sugar, which then causes adipose tissue and the liver to break down glycogen and triglycerides, which in return gives us a reduction in total body fat and a reduction in energy
consumption for its breakdown. IGF1-LR3 reduces insulin levels as well as insulin requirements in patients with type 1 diabetes. In most cases this translates into a 10% reduction in insulin requirements in order to maintain the same blood sugar level.
IGF1-LR3 and other IGF-1 derivatives are able to counteract the negative effects of myostatin to protect muscle cells and prevent apoptosis. IGF1-LR3, thanks to its long half-life, does quite well at inhibiting myostatin and seems to work by activating a muscle protein called MyoD. MyoD is a protein activated by exercise or tissue damage and is responsible for muscle hypertrophy.
Looking at it from an anti-aging side, IGF1-LR3 promotes the repair and health of all tissues in our body (cow and pig studies) ongoing mice studies for the prevention of senile diseases such as dementia, muscle wasting and kidney disease.
Although IGF-1 is very similar to insulin (hence the name Insulin-like growth factor 1), its role is slightly different. Like insulin, IGF-1 is a transport hormone that helps nutrients (amino acids and glucose) reach the muscle cells. The cells themselves can then use these ingredients to synthesize new muscle tissue.
IGF-1 also works anabolic in bone, connective and intestinal tissues. This differs from insulin, which is a nutrient-transporting hormone in the broader sense of the word, because insulin transports nutrients not only to muscle tissue but also to many
other tissues throughout the body, so looking at it from a refined coke, insulin is not not selective at all, and this is what selectivity is all about.
Furthermore, IGF-1 plays a very unique and very specific number of roles in the human body, and this changes at every stage of human development. For example, it is responsible for various important factors of growth during childhood, and is also anabolic in adults. From a medical point of view, its use is mainly for the treatment of growth disorders, but since IGF-1 is a relatively new development in medicine, its experimental use in the treatment of other conditions continues to expand. Clinical research and application include, but are not limited to:
Dwarfism
Anti-aging
Neuropathy
Cancer
Stroke
There are many variants of IGF-1, IGF-1, IGF-1 LR3 and IGF-1 DES, and they all have different half-life and use-life properties that are associated with this period. For IGF-1 DES, the dosage range is 50 to 150 mcg per day. Due to a much shorter half-life than the LR3 variant, higher doses can be used without greater risk of long-term effects on the body, although caution should still be exercised.
MGF (PEG) Mechano Growth Factor - a mechanical growth factor
Also known as PEG-MGF, this peptide not only promotes muscle growth but also causes the formation of new muscle cells. IGF-1 is bound to polyethylene glycol (PEG) which distinguishes it from common mechanical growth factor. It follows that the MGF
half-life increases from minutes to several days. It is better to use PEG-MGF after training. The reason for this is the mechanical-sensitive nature of the MGF. This means that it is activated by a mechanical stimulus. The peptide facilitates faster recovery from muscle damage.
The list of the peptides mentioned is, of course, not exhaustive. There are many other types on the market, some of which are not necessarily beneficial for physique sports.
Peptides are dosed per weight, can (and even should) be mixed (GHRH + GHRP) when we don't have to be careful when natural burst is happening to get the best out of them.
It should be emphasized here that the amount of growth hormone released depends on what peptides we connect. An example of a combination that can provide very good results is GHRP-2 and MOD-GRF (1 - 29). It has been clinically proven that 100 mcg of both will result in a HGH burst comparable to more than 7iu in pharmacy hGH.
BPC - 157 (Body Protecting Compound)
L-Valine, glycyl-L-alpha-glutamyl-L-prolyl-L-prolyl-L- prolylglycy l-L-lysyl-L-prolyl-L-alanyl-L-alpha-aspartyl-L-alpha-
aspartyl-L-alanylglycyl-L-leucyl-; glycyl-L-alpha-glutamyl-L- prolyl-L-prolyl-L-prolylglycyllysyl-L-prolyl-L-alanyl-L-alpha- aspartyl-L-alpha-aspartyl-L-alanylglycyl-L-leucyl-L-valine, i jak widać jest łańcuch 15 aminokwasów.
As with all peptides, it should be handled carefully, saline should be poured down the side of the bottle, and if bacteriostatic water is available, it is preferable because then we do not need to keep it in the refrigerator and there will be more space for insulin and hGH, which We do not shake the solution in the bottle more and we handle it like an egg. The sufficient dosage, on which
I felt the effect of relief on the shoulder, was 250 mcg (I inserted it between the clavicle bone and the front akton, i.e. as close as possible to the place of the alleged injury, subcutaneously). It can be administered orally (we hold it in the mouth for about 2 minutes then swallow) or intramuscularly - here it will be known
- more painful, but where the joy of the injection coating and the peptide is, anyway, gets to the place where it is greatest demand.
Let's start with the fact that it is a measure not wrapped by a ban by WADA (at the moment!), It is not subject to any patents, so pharmaceutical companies cannot earn on it, and it is cheap as water, a lot of research that it works and is legal but let's get to the heart of the matter.
Our body produces BPC in very small amounts in gastric juices, where it serves to protect and "heal" the intestines, but when we grab a concentrated product, e.g. from BioLab, Peptides UK, it will have a very high level biological activity and theoretically (and I have already found out in practice) no matter where we stick it, it will start
repairing damaged tissues - all tissues
Teeth
Muscles
Bowels
Bones
Tendons
What has been proven not only in rats or IVF, but also in humans.
BPC also counteracts the side effects of non-steroidal inflammatory drugs such as diclofenac, naproxen,/22950504)
BPC is stable in gastric juice, it can induce an anabolic healing effect in the upper and lower gastrointestinal tract, has anti-ulcer properties and has a very good therapeutic effect against IBD, all without any side effects? Yeah buddy!
As proven in studies, BPC accelerates wound healing and, through interaction with nitric oxide, protects the endothelial tissues as well as forces increased angiogenze (for the less inquisitive - the process of capillary formation). Even in very poorly favorable conditions for regeneration, such as irritable bowel syndrome - during this action, thanks to BPC, the expression of genes responsible for cytokines and the production of growth factor, as well as the extracellular matrix,
i.e. collagen, is stimulated. Add to this the treatment of such problems as, for example, pathological anastomoses, and in this case - intestinal-intestinal fistulas (i.e. intestinal loop anastomosis), the administration of the BPC 157 peptide showed properties equal to between 15 - 40 mg of such antidepressants as, for example, imipramine - the first fully effective an antidepressant drug, used in 1957. It inhibits the reuptake of norepinephrine and serotonin from the synaptic cleft. It has a moderate peripheral and central anti-cholinergic effect and a weak antihistamine effect. 50% is absorbed from the gastrointestinal tract, metabolized in the liver to desipramine, an active metabolite that also has a psychotropic effect.
In tests with drugs from the diazepam group, such as e.g. valium BPC-157, it showed the following properties
Decreased tolerance to diazepam - lower dosage of drug
Reduced drug "withdrawal" symptoms
Anticonvulsant efficacy
Theoretically - the administration of this peptide simultaneously with diazepam may act postsynaptically, preventing the reduction of the level of benzodiazepine receptors, i.e. simply and to the point - it can be speculated that BPC157 has a beneficial effect on the natural homeostasis of the GABA receptor complex, and also strengthens GABAergic transmissions.
It has been known for a long time that BPC157 has a positive protective effect on the gastrointestinal tract and other internal organs (e.g. the heart), but it has also recently been proven that it has a protective effect on the dopaminergic system (in rats).
Blocks the side effects and motor stereotypes induced
by methamphetamine abuse
It interacts fully with the dopamine system - centrally and peripherally
It prevents certain stages of catalepsy (the symptom is caused by an increase in muscle tone with a simultaneous impairment or even complete blockage of the patient's motor functions. This stiffening is flexible, i.e. the
patient's body can be moved) and the formation of ulceration.